By S. J. Haught
Writer: S.J. Haught
Paperback: one hundred thirty five pages
Publisher: Gerson Institute
S.J. Haught interviewed Dr. Gerson for his tale, "The Unveiling of a Quack." in its place he wrote a narrative that he describes as approximately "a guy who by way of absolute checklist had cured humans of melanoma, together with young children, and his really brave and lonely struggle opposed to the forces of equipped medicine."
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Additional info for Censured for Curing Cancer: The American Experience of Dr. Max Gerson
2004). As only 5–10 per cent of all smokers develop lung cancer, there has been considerable interest in identifying genetic factors that might predispose to lung cancer by increasing susceptibility to environmental carcinogens. Genetic polymorphisms in the activation or detoxiﬁcation of carcinogens (such as those in tobacco smoke) could be associated with interindividual susceptibilities to lung cancer. g. g. GSTM1 and GSTT1). , 2003). , 2002). Although signiﬁcant associations have been reported, these have not always replicated (see Hirvonen, 1995).
1996b). The aetiology of sporadic MTC is also not well known. Amongst all presentations of MTC, clinical epidemiologic studies have shown that 25 per cent can be attributable to MEN 2. , 1997). Due to these accumulating data, the general recommendations are to offer RET mutation analysis to all presentations of MTC irregardless of age, presence of syndromic features or family history. , 1996a). The great majority of somatic RET mutations are M918T. , 1996b; 1998). , 1996a). It remains controversial whether somatic M918T in the primary tumour portends a poor prognosis.
14 per 100 000 and for the most part, occurs as part of the multiple endocrine neoplasia syndromes (MEN; see p. 220 and 222), with chief cell hyperplasia as the usual histologic change. The genetic differential diagnosis of parathyroid hyperplasia or parathyroid adenoma includes MEN 1, caused by germline MEN1 mutations; MEN 2, caused by germline RET mutations; and hyperparathyroidism jaw tumour syndrome (HPT-JT), caused by germline mutations in HRPT2. , 2000). , 1993). , 1998). , 1996a). , 2002).